↵ * If one of the first 3 criteria is not met, a diagnosis of definite limbic encephalitis can be made only with the detection of antibodies against cell-surface, synaptic, or onconeural proteins.. ↵ ...

In most autoimmune encephalitides, the MRI is normal or shows nonspecific inflammatory changes; two exceptions are limbic encephalitis and anti-GABA A R encephalitis.

MRI imaging of the brain is usually nonspecific, or even normal, but limbic encephalitis usually produces some characteristic changes in the medial temporal lobe.

Diagnosis Voltage-gated potassium channel antibody-related limbic encephalitis. Management Antiepileptic drugs, immunomodulatory therapy, oral steroids, plasma exchange.

Conclusions . Paraneoplastic anti-NMDA receptor encephalitis is potentially lethal, but usually reversible if promptly recognized and treated. It should be suspected in young women with prominent ...

MRI: abnormal findings by conventional MRI sequences Abnormal intensity in limbic cortices, frontal and temporal lobes (hyperintense on FLAIR and T2-W, hypointense on T1-W) ...

Schäfer J, Christensen PB, Jensen K. AMPA and NMDA receptor antibody autoimmune encephalitis preceded by ocular myasthenia gravis: a case report. BMC Neurol . Published online March 10, 2023. doi ...

MRI features suggestive of encephalitis, including T 2 /fluid-attenuated inversion recovery (FLAIR) hyperintensity highly restricted to one or both medial temporal lobes Reasonable exclusion of ...

MRI findings show findings of limbic encephalitis, such as T2-FLAIR abnormalities in the medial half of the temporal lobe. CASPR2 antibody-associated encephalitis ...